Structure and calcium-binding studies of calmodulin-like domain of human non-muscle α-actinin-1

نویسندگان

  • Sara Drmota Prebil
  • Urška Slapšak
  • Miha Pavšič
  • Gregor Ilc
  • Vid Puž
  • Euripedes de Almeida Ribeiro
  • Dorothea Anrather
  • Markus Hartl
  • Lars Backman
  • Janez Plavec
  • Brigita Lenarčič
  • Kristina Djinović-Carugo
چکیده

The activity of several cytosolic proteins critically depends on the concentration of calcium ions. One important intracellular calcium-sensing protein is α-actinin-1, the major actin crosslinking protein in focal adhesions and stress fibers. The actin crosslinking activity of α-actinin-1 has been proposed to be negatively regulated by calcium, but the underlying molecular mechanisms are poorly understood. To address this, we determined the first high-resolution NMR structure of its functional calmodulin-like domain (CaMD) in calcium-bound and calcium-free form. These structures reveal that in the absence of calcium, CaMD displays a conformationally flexible ensemble that undergoes a structural change upon calcium binding, leading to limited rotation of the N- and C-terminal lobes around the connecting linker and consequent stabilization of the calcium-loaded structure. Mutagenesis experiments, coupled with mass-spectrometry and isothermal calorimetry data designed to validate the calcium binding stoichiometry and binding site, showed that human non-muscle α-actinin-1 binds a single calcium ion within the N-terminal lobe. Finally, based on our structural data and analogy with other α-actinins, we provide a structural model of regulation of the actin crosslinking activity of α-actinin-1 where calcium induced structural stabilisation causes fastening of the juxtaposed actin binding domain, leading to impaired capacity to crosslink actin.

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عنوان ژورنال:

دوره 6  شماره 

صفحات  -

تاریخ انتشار 2016